https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:54887 Wed 20 Mar 2024 13:18:21 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27875 Wed 11 Apr 2018 10:34:23 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27387 14.3 years. After adjusting for confounders, diabetic subjects were 49% (cause-specific hazard ratio (_cHR) = 1.49, 95% CI: 1.37, 1.62) more likely to develop kidney failure than non-diabetic subjects. Albuminuria categories A3 and A2 were, respectively, 3.40 (95% CI: 3.07, 3.76) and 1.71 (95% CI: 1.53, 1.92) higher risk of kidney failure when compared to A1. For each albumin category, death rate increased as albuminuria increased particularly in diabetic subjects, which was approximately 2 times higher in A3 compared to A1. Considering GFR category, it gradually increased from G1 to G4 and sharply increased from G4 to G5 in both non-diabetic and diabetic subjects. This study has quantified CKD progression in an Asian population within ordinary practice. Diabetic subjects progress through GFR and albuminuria categories and reach kidney failure about twice as rapidly as non-diabetic subjects.]]> Wed 11 Apr 2018 10:07:38 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:11389 Sat 24 Mar 2018 08:10:00 AEDT ]]>